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353

It only kills you in 10 years now. Not 2.

Archive: https://archive.today/embmS

From the post:

>Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), responsible for the COVID-19 pandemic, utilizes membrane-bound, angiotensin-converting enzyme II (ACE2) for internalization and infection. We describe the development of a biologic that takes advantage of the proximity of the N-terminus of bound ACE2 to the three-fold symmetry axis of the spike protein to create an ultrapotent, trivalent ACE2 entry antagonist. Distinct disulfide bonds were added to enhance serum stability and a single point mutation was introduced to eliminate enzymatic activity. Through surface plasmon resonance, pseudovirus neutralization assays, and single-particle cryo-electron microscopy, we show this antagonist binds to and inhibits SARS-CoV-2 variants. We further show the antagonist binds to and inhibits a 2003 SARS-CoV-1 strain. Collectively, structural insight has allowed us to design a universal trivalent antagonist against all variants of SARS-CoV-2 tested, suggesting it will be active against the emergence of future mutants.

It only kills you in 10 years now. Not 2. Archive: https://archive.today/embmS From the post: >>Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), responsible for the COVID-19 pandemic, utilizes membrane-bound, angiotensin-converting enzyme II (ACE2) for internalization and infection. We describe the development of a biologic that takes advantage of the proximity of the N-terminus of bound ACE2 to the three-fold symmetry axis of the spike protein to create an ultrapotent, trivalent ACE2 entry antagonist. Distinct disulfide bonds were added to enhance serum stability and a single point mutation was introduced to eliminate enzymatic activity. Through surface plasmon resonance, pseudovirus neutralization assays, and single-particle cryo-electron microscopy, we show this antagonist binds to and inhibits SARS-CoV-2 variants. We further show the antagonist binds to and inhibits a 2003 SARS-CoV-1 strain. Collectively, structural insight has allowed us to design a universal trivalent antagonist against all variants of SARS-CoV-2 tested, suggesting it will be active against the emergence of future mutants.

(post is archived)

[–] 2 pts (edited )

Genuflect for publication...

Remarkably, vaccines were produced in record time and are now widely available, providing protection and dramatically reducing death rates

Take a little swipe at the science deniers...

Despite these efforts, the virus continues to spread for numerous reasons, including variable immune responses and vaccine hesitancy

Not feeling warm and fuzzy, but I will read on...

Furthermore, as both SARS-CoV-1 and SARS-CoV-2 likely originated from bats

Fuck. Time to look at author names... Yead looks suspicious but xe is in the middle. Corresponding author is Gonzales and could break either way.

So it is not proposing genetic bullshit yet, they are designing a protein to disrupt ace2 and spike interaction. They are spamming the virus. I'll be interested in the next step.