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571

It only kills you in 10 years now. Not 2.

Archive: https://archive.today/embmS

From the post:

>Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), responsible for the COVID-19 pandemic, utilizes membrane-bound, angiotensin-converting enzyme II (ACE2) for internalization and infection. We describe the development of a biologic that takes advantage of the proximity of the N-terminus of bound ACE2 to the three-fold symmetry axis of the spike protein to create an ultrapotent, trivalent ACE2 entry antagonist. Distinct disulfide bonds were added to enhance serum stability and a single point mutation was introduced to eliminate enzymatic activity. Through surface plasmon resonance, pseudovirus neutralization assays, and single-particle cryo-electron microscopy, we show this antagonist binds to and inhibits SARS-CoV-2 variants. We further show the antagonist binds to and inhibits a 2003 SARS-CoV-1 strain. Collectively, structural insight has allowed us to design a universal trivalent antagonist against all variants of SARS-CoV-2 tested, suggesting it will be active against the emergence of future mutants.

It only kills you in 10 years now. Not 2. Archive: https://archive.today/embmS From the post: >>Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), responsible for the COVID-19 pandemic, utilizes membrane-bound, angiotensin-converting enzyme II (ACE2) for internalization and infection. We describe the development of a biologic that takes advantage of the proximity of the N-terminus of bound ACE2 to the three-fold symmetry axis of the spike protein to create an ultrapotent, trivalent ACE2 entry antagonist. Distinct disulfide bonds were added to enhance serum stability and a single point mutation was introduced to eliminate enzymatic activity. Through surface plasmon resonance, pseudovirus neutralization assays, and single-particle cryo-electron microscopy, we show this antagonist binds to and inhibits SARS-CoV-2 variants. We further show the antagonist binds to and inhibits a 2003 SARS-CoV-1 strain. Collectively, structural insight has allowed us to design a universal trivalent antagonist against all variants of SARS-CoV-2 tested, suggesting it will be active against the emergence of future mutants.

(post is archived)

[–] • 2 pts

From what im reading here this doesn't require you to reprogram your genes with an mRNA jab you just need to get the Antagonist into your bloodstream or the infection site, probably both, prevent the spike protein from binding to your cell walls. In fact this actually looks like it would solve almost all of the problems (of a chronic nature) of taking the mRNA jabs. The antagonist binds to spike protein in such a way that it cant bind with other things meaning it can't open your cells. Meaning covid cant reproduce if the agonist binds to it and loose spike protein from being genetically modified can no longer bind to cells either. Existing damage wont be fixed but new damage would halt so long as you have this antagonist in serum in equal or extraneous proportion to the spike protein in your blood.

There is still the problem of clutter which will cause clotting but it may prevent spike protein felting which causes the fibrous clotting.

Clot shot enjoyers are still fucked they'll be paying for these meds for life and still have complications. The antagonist appears to be sulfur based so it seems they could make an inhalent that us as safe or safer than albuterol that, taken over a few days would cure any SARS variant and lesson symptoms dramatically.

Ironically, if this isn't implemented as mRNA it will represent a revolutionary advancement in anti viral medicine.

TL;DR treating viruses in this fashion may lead to a world where curing a viral infection is as straightforward as administering an anti-venom for a spider or snake bite.

The reality is this solution is essentially the solution your immune system likely developed to treat covid after catching it because it's the same category of solution your immune system uses to solve any virus. And that explains why such a huge proportion of retards who got the clot shot and didn't die, haven't died yet, they caught covid developed an antigen that behaves like this spike protein antagonist that plugs the spike protein making it inoperable and have been pumping that antigen out constantly for years now. Like I said, they still have trash cluttering up their circulatory system so could still die quite a bit earlier than purebloods.

[–] • 2 pts (edited )

Genuflect for publication...

Remarkably, vaccines were produced in record time and are now widely available, providing protection and dramatically reducing death rates

Take a little swipe at the science deniers...

Despite these efforts, the virus continues to spread for numerous reasons, including variable immune responses and vaccine hesitancy

Not feeling warm and fuzzy, but I will read on...

Furthermore, as both SARS-CoV-1 and SARS-CoV-2 likely originated from bats

Fuck. Time to look at author names... Yead looks suspicious but xe is in the middle. Corresponding author is Gonzales and could break either way.

So it is not proposing genetic bullshit yet, they are designing a protein to disrupt ace2 and spike interaction. They are spamming the virus. I'll be interested in the next step.