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source: https://www.technocracy.news/dr-michael-yeadon-not-one-of-those-things-is-supported-by-the-science/

Author: Michael Yeadon, Ph.D., a life science researcher and former vice-president and chief scientist of allergy and respiratory research at Pfizer

“Viruses are really tiny, and their business is to get as quickly as they can inside your cells. So, they bind to a receptor on the surface and inject themselves into your cell. So, they’re inside. Antibodies are big molecules and they’re generally outside your cells.

So just think about that for a moment. Antibodies and viruses are in separate compartments. The virus is inside the cell, the antibodies outside the cell. I’m not saying antibodies have no role, but they’re really not very important. This has been proven. There are some people in whom a natural experiment has occurred.

They have a defect and they actually don’t make antibodies, but they’re able to fight off COVID-19, the virus SARS-CoV-2, quite well. The way they do that is, they have T-cell immunity, cellular immunity. [T-cells] are cells that are trained to detect virus-infected cells and to kill those cells. That’s how you defend yourself against a virus.

So, all of these mentions of antibody levels, it’s just bunk. It is not a good measure of whether or not you’re immune. It does give evidence that you’ve been infected, but their persistence is not important as to whether you’ve got immunity …

We’ve known this for decades. We’ve known about T-cells for decades. They were clearly in my undergraduate textbooks. And we’ve known about their importance in defending you against respiratory viruses since probably the 1970s, certainly the 1980s. So, don’t believe anything where people suggest to you that their role is uncertain. We’ve known for a very long time that they are absolutely central.”

source: https://www.technocracy.news/dr-michael-yeadon-not-one-of-those-things-is-supported-by-the-science/ Author: Michael Yeadon, Ph.D., a life science researcher and former vice-president and chief scientist of allergy and respiratory research at Pfizer >“Viruses are really tiny, and their business is to get as quickly as they can inside your cells. So, they bind to a receptor on the surface and inject themselves into your cell. So, they’re inside. Antibodies are big molecules and they’re generally outside your cells. > So just think about that for a moment. Antibodies and viruses are in separate compartments. The virus is inside the cell, the antibodies outside the cell. I’m not saying antibodies have no role, but they’re really not very important. This has been proven. There are some people in whom a natural experiment has occurred. > They have a defect and they actually don’t make antibodies, but they’re able to fight off COVID-19, the virus SARS-CoV-2, quite well. The way they do that is, they have T-cell immunity, cellular immunity. [T-cells] are cells that are trained to detect virus-infected cells and to kill those cells. That’s how you defend yourself against a virus. > So, all of these mentions of antibody levels, it’s just bunk. It is not a good measure of whether or not you’re immune. It does give evidence that you’ve been infected, but their persistence is not important as to whether you’ve got immunity … > We’ve known this for decades. We’ve known about T-cells for decades. They were clearly in my undergraduate textbooks. And we’ve known about their importance in defending you against respiratory viruses since probably the 1970s, certainly the 1980s. So, don’t believe anything where people suggest to you that their role is uncertain. We’ve known for a very long time that they are absolutely central.”

(post is archived)

[–] 1 pt (edited )

http://www.youtube.com/watch?v=Y2QNnoqc3XU

Is the scene from a 2020 movie(edit Tv show, Utopia remake)(clearly predictive programming giving the circumstances right now)

You clearly know more about infections then I do, but I'm very good at sniffing out things...I'll try to offer a hint that you could perhapse elaborate on given your field of study or just being smart at that particular subject

As I understand it (forgive laymens terms) the S spike protein is VERY similar in "shape" i guess you'd say, to a protein that either is fully, or largely responsible, for the adhering of the placental tissue to the uteran(sp) wall. This "vaccine" will cause women's bodies to identify any protein closely resembling the spike protein in "covid" and cause the body to attack it.

What i suspect is that there is no actual corona, and this "spike protein" is pushed as the "solution" of infection because of it's closely resembelling the placental protein. This will allow them to, once infertility starts becoming openly noticed, say "Oh, look at that, a side effect of the vaccine, ah darn, we didn't know" or something along those lines...I doubt whoever had a hand in this doesn't have some sort of plan where they stay in power despite what they just did.

So basically I think they are using the cover of a fake epidemic to get people to rush out and beg to be sterilized (reality)

but instead they think they are rushing out to do their part to protect humanity

it's like...satan himself is running a bait and switch operation...

[–] 0 pt

You are probably right and if no one believes you, then tell them to read this study that explains in detail how blocked ACE2 affects egg meoisis and spermatogenesis as well as other subtle metabolic disorders to slow you down.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3821659/#!po=35.7143