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source: https://www.technocracy.news/dr-michael-yeadon-not-one-of-those-things-is-supported-by-the-science/

Author: Michael Yeadon, Ph.D., a life science researcher and former vice-president and chief scientist of allergy and respiratory research at Pfizer

“Viruses are really tiny, and their business is to get as quickly as they can inside your cells. So, they bind to a receptor on the surface and inject themselves into your cell. So, they’re inside. Antibodies are big molecules and they’re generally outside your cells.

So just think about that for a moment. Antibodies and viruses are in separate compartments. The virus is inside the cell, the antibodies outside the cell. I’m not saying antibodies have no role, but they’re really not very important. This has been proven. There are some people in whom a natural experiment has occurred.

They have a defect and they actually don’t make antibodies, but they’re able to fight off COVID-19, the virus SARS-CoV-2, quite well. The way they do that is, they have T-cell immunity, cellular immunity. [T-cells] are cells that are trained to detect virus-infected cells and to kill those cells. That’s how you defend yourself against a virus.

So, all of these mentions of antibody levels, it’s just bunk. It is not a good measure of whether or not you’re immune. It does give evidence that you’ve been infected, but their persistence is not important as to whether you’ve got immunity …

We’ve known this for decades. We’ve known about T-cells for decades. They were clearly in my undergraduate textbooks. And we’ve known about their importance in defending you against respiratory viruses since probably the 1970s, certainly the 1980s. So, don’t believe anything where people suggest to you that their role is uncertain. We’ve known for a very long time that they are absolutely central.”

source: https://www.technocracy.news/dr-michael-yeadon-not-one-of-those-things-is-supported-by-the-science/ Author: Michael Yeadon, Ph.D., a life science researcher and former vice-president and chief scientist of allergy and respiratory research at Pfizer >“Viruses are really tiny, and their business is to get as quickly as they can inside your cells. So, they bind to a receptor on the surface and inject themselves into your cell. So, they’re inside. Antibodies are big molecules and they’re generally outside your cells. > So just think about that for a moment. Antibodies and viruses are in separate compartments. The virus is inside the cell, the antibodies outside the cell. I’m not saying antibodies have no role, but they’re really not very important. This has been proven. There are some people in whom a natural experiment has occurred. > They have a defect and they actually don’t make antibodies, but they’re able to fight off COVID-19, the virus SARS-CoV-2, quite well. The way they do that is, they have T-cell immunity, cellular immunity. [T-cells] are cells that are trained to detect virus-infected cells and to kill those cells. That’s how you defend yourself against a virus. > So, all of these mentions of antibody levels, it’s just bunk. It is not a good measure of whether or not you’re immune. It does give evidence that you’ve been infected, but their persistence is not important as to whether you’ve got immunity … > We’ve known this for decades. We’ve known about T-cells for decades. They were clearly in my undergraduate textbooks. And we’ve known about their importance in defending you against respiratory viruses since probably the 1970s, certainly the 1980s. So, don’t believe anything where people suggest to you that their role is uncertain. We’ve known for a very long time that they are absolutely central.”

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[–] • 2 pts

The spike protein binds to your cells, like the Dr. said. The specific receptor that the spike protein binds to is called ACE2, which stands for angiotensin converting enzyme 2. As the name implies it's an enzyme receptor which converts the hormonal protein angiotensin 2.

Angiotensin 2 is a hormone that regulates some body functions, which regulates other hormones, which regulates some other body functions.

When the spike protein binds to ACE2 receptors in order to infect you with the virus, it also has the effect of stopping the ACE2 receptors from breaking down angiotensin 2. Because of this, the serum level of angiotensin 2 increases in the blood, increasing vasopressin and aldosterone production. The more spike proteins are in your body, the higher the probability that the proteins are affecting ACE2 function, leading to a hormonal imbalance.

I'll let you figure out what happens when you have extreme hormonal imbalances, when you have angiotensin 2 poisoning or excess aldosterone or vasopressin.

[–] • 2 pts

Excellent summary. Have you read Salk Institute report?

[–] • 0 pt

Link?

[–] • 1 pt

https://www.ahajournals.org/doi/10.1161/CIRCRESAHA.121.318902

I'm impressed you came to your conclusion prior to reading this. Also, thanks for summarizing in a readily understandable format. I've saved it and will likely plagiarize it.

https://www.salk.edu/news-release/the-novel-coronavirus-spike-protein-plays-additional-key-role-in-illness/

[–] • 2 pts

Btw, thanks. Will do some more reading on this. I suck at organic chemistry.

[–] • 2 pts

It is also an extension of physiology too. ACE receptors and renin angiotensin aldosterone system are involved in blood pressure regulation. At least three different hypertension medications exist strictly to inhibit this RAAS system in various ways.

If this gene therapy imbalances this system to increase concentrations of associated molecules, then you can expect insane hypertensive episodes. Given how many people are clotting and stroking out, it is within the realm of possibility that it is being triggered by the damage to the vascular system caused by hypertension.

[–] • 2 pts

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3891217/

Once again comes back to fertility.

[–] • 1 pt

When you understand where the virus attacks you and what it's interrupting it kind of makes sense, and if this was genetically engineered, they wanted this to be quite nasty. Luckily for us they're fucking morons.

[–] • 2 pts

So either it escaped wuhan before it was ready for prime time -or- an intentionally less deadly variant was used to frame CCP. Seems obvious.