*I found this post on another site and wanted to share it...* * Amygdalin induces apoptosis in human cervical cancer cell line HeLa cells: https://pubmed.ncbi.nlm.nih.gov/23137229/ * Amygdalin Blocks Bladder Cancer Cell Growth In Vitro by Diminishing Cyclin A and cdk2: https://journals.plos.org/plosone/article?id=10.1371%2Fjournal.pone.0105590 * Amygdalin Influences Bladder Cancer Cell Adhesion and Invasion In Vitro: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4198254/ * Amygdalin Regulates Apoptosis and Adhesion in Hs578T Triple-Negative Breast Cancer Cells: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4703354/ * Amygdalin Induces Apoptosis through Regulation of Bax and Bcl-2 Expressions in Human DU145 and LNCaP Prostate Cancer https://www.jstage.jst.go.jp/article/bpb/29/8/29_8_1597/_pdf * Amygdalin inhibits genes related to cell cycle in SNU-C4 human colon cancer cells: https://pubmed.ncbi.nlm.nih.gov/16127745/ * Apoptosis induction of Persicae Semen extract in human promyelocytic leukemia (HL-60) cells: https://pubmed.ncbi.nlm.nih.gov/12643594/ * Enhancement of amygdalin activated with β-d-glucosidase on HepG2 cells proliferation and apoptosis (Liver): https://www.sciencedirect.com/science/article/abs/pii/S0144861712005322 * Amygdalin-mediated inhibition of non-small cell lung cancer cell invasion in vitro: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4503109/ Here are the best dietary sources of B-17. https://optinghealth.com/foods-high-in-vitamin-b17/ **This is a sample of the text from the prostate cancer paper attached above from 2006:** *Many hypotheses have been proposed to explain the anti- cancer effects of amygdalin. For instance, amygdalin may be specifically broken down by beta-glucosidase, which is abun- dant in cancer cells, and consequently cyanide is released onto the cancerous lesions wherein it exerts toxicity on the cancer cells.37—39) Another suggestion is that amygdalin en- hances the functions of pancreatic enzymes, which may pre- vent transformation of cancer primordial germ cells.4,38) A third hypothesis is that amygdalin (vitamin B17) restores the vitamin deficiency that could lead to metabolic disorders in cancer patients.40) In particular, Bhatti et al.41) reported that amygdalin may stimulate the immune system to produce anti-cancer activity in prostate cancer patients. In the present study, we demonstrated that amygdalin in- duces apoptotic cell death by caspase-3 activation through the down-regulation of anti-apoptotic Bcl-2 protein and the up-regulation of pro-apoptotic Bax protein in DU145 and LNCaP prostate cancer cells. Based on these results, amyg- dalin shows considerable promise in the treatment of prostate cancer.*