Perhaps you can clarify where I'm wrong, but what you linked is not a very convincing argument that any sar-cov-2 has been isolated and proven to cause illness.
The study is two separate cases of people suspected of having Sars-cov-2. And the claim is that both were found to have genetically identical cov viruses that correspond to sar-cov-2 in them. Quoting what they did with patient 2:
"A conventional nested RT-PCR test of this patient was positive for the CoV family 9. Subsequent genome sequencing was again compatible with the SARS-CoV-2 and shared via the GISAID EpiCoV database (EPI_ISL_403963). A nasopharyngeal swab also tested positive by RT-PCR for adenovirus."
Does this mean they took her sample, ran it through a PCR to amplify the material, and then tested to see if there was anything significantly similar to known CoV viruses? They found Cov virus and also found adenovirus? And then they tested to see if the sample was "compatible" with the Sars-cov-2 genome that they claim to know.
Couldn't you easily find CoV viruses in a healthy person's sample if you ran it through PCR? Aren't viruses everywhere? Aren't people covered in them? Aren't CoV viruses extremely common? Wouldn't we expect everyone to have them, especially after PCR?
Then the "known" sars-cov-2 is shown to also be "compatible" with the samples. But were they working with the isolated cov virus?
If sar-cov-2's "known" code is made of sequences that are found in a normal human sample, and you are not testing the isolated virus and are instead testing a not-isolated sample, you could find all the components of sar-cov-2's "known" sample even though there is no actual occurrence of all of the sequences inside a virus. This seems to be the trouble to people like myself - they're never actually isolating out the virus, they're always just genetic sequencing contaminated samples and cherrypicking what they want.
I'm no expert, but to me it appears that the scientists in this field are playing genetic games on their "illumina" computer (which the study used), but they aren't ever actually replicating the virus in host cells or proving that any specific virus is causing illness. Has PCR testing made scientists lazy? When you've seen the utter lies and trickery that climate scientist have gotten away with with their computer models, you'll forgive some skepticism for in-silico research.
The first link just shows what they have done to find out if two patients have something still unknown in common - for people who are interested in how a group of scientists describe their work so that other groups can poke holes in it. Maybe you have found a big hole in their work, but they were not forced by colleagues to retract their paper yet.
what you linked is not a very convincing argument that any sar-cov-2 has been isolated [..]
The second link proves at least one thing: Isolation is routine work. For example, done a thousand times for the new influenza A/H3N2 strain. Done for decades. Done worldwide (not only by western pharma slaves). Done 3.4 million times for SARS-CoV-2 alone, maybe a billion times for all other viruses together.
You may find a hole in this process, making scientists very unhappy. But then you have found that scientific research on all viruses was based on wrong assumptions. This is what the terrain theory people do. But to say that science did it right until December 2019 and then suddenly got it all wrong doesn't make sense. Except if you assume that they all conspire, but keep in mind that then scientists from 190 countries must collaborate.
what you linked is not a very convincing argument that any sar-cov-2 has been [..] proven to cause illness.
They can show that humanized mice get symptoms when infected with the isolated virus. Humanized means that the genes are changed in a way that the mice got human features like human ACE2 receptors. An even better proof would be to use soldiers or Uighurs. The US government has a long history of using their own soldiers for such experiments, maybe the Chinese do it in their camps - but they don't release the results.
Another proof is indirect: 3.4 million samples of 30k nucleotides long RNA that differ in just 12 regions were extracted from symptomatic patients.
In the end, scientists are happy with very indirect proofs. For example, the extracted RNA encodes a protein that binds to ACE2 (proven with cell cultures that were modified to have more or less ACE2), the protein needs to get split into two parts so that one part can merge with the cell membrane to get sucked into the cell (and with it the virus, a theory made in silico), and if the molecules needed to do the splitting get blocked, the virus cannot enter the cell (proven in vitro), which led to trials with one blocker that showed that giving bromhexine (OTC in Europe) cuts the death rate in half.
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