Yes.
https://www.jci.org/articles/view/96702
As a result of its unique metabolic properties, the fructose component of sugar may be particularly harmful. Diets high in fructose can rapidly produce all of the key features of the metabolic syndrome.
https://www.mdpi.com/2072-6643/9/3/230/pdf
excessive fructose consumption is known to favor nonalcoholic fatty liver disease (NAFLD), as fructose is both a substrate and an inducer of hepatic de novo lipogenesis.
https://www.academia.edu/download/86268272/j.orcp.2012.11.00220220522-1-1159in9.pdf
Fructose is a monosaccharide and reducing sugar. It is present in sucrose and honey. Researchers around the world have come together in a just-published study that offers new ideas about how fructose consumption results in obesity and metabolic syndrome, which can lead to diabetes. In this review, we discuss that how fructose causes fatty liver, obesity and insulin resistance. We also discuss the effects of consumption of high fructose corn syrup, dietary fructose, fructose-induced changes in metabolism.
Want more? There are dozens.
Excess anything.
There's a distinct difference between fruit and sugar.
Excess anything.
Excess anything doesn't cause metabolic disorders like fatty liver disease, diabetes, and dyslipidemia. Only fructose does that, and "excess" fructose means that 5 apples, or three prunes will put you over the top.
There's a distinct difference between fruit and sugar.
Fruit contains sugar, fructose specifically. It takes very little of some fruits to damage your body.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2673878/
We have demonstrated that a mechanism by which fructose induces postprandial hypertriglyceridemia is through increased hepatic de novo lipogenesis. It has long been established that in contrast to the metabolism of glucose, fructose metabolism is independent of phosphofructose kinase regulation; thus, its uptake by the liver and its metabolism to de novo lipogenesis substrate is not limited by energy status (cytosolic ATP and citrate levels). In addition, fructose may activate sterol receptor element–binding protein-1c independently of insulin, which activates genes involved in de novo lipogenesis.
You're literally a retard.
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