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Looking for research papers on ADE deaths during development of mRNA vaccines. I have seen several posts claiming that research animals in mRNA vaccine studies all died from ADE, but I cannot find any of those papers. Do any of you have them or know where I might be able to find them?

Looking for research papers on ADE deaths during development of mRNA vaccines. I have seen several posts claiming that research animals in mRNA vaccine studies all died from ADE, but I cannot find any of those papers. Do any of you have them or know where I might be able to find them?

(post is archived)

[–] • 9 pts (edited )

Used the search on poal that anyone* can use (magnifier by your messages icon) and searched for "trials": *(anyone who regularly participates on the site to get beyond 'new account' levels)

3rd result from 6 days ago by is very good:

A reddit post about previous mRNA injection attempts where the trials made things worse. https://poal.co/s/Vax/427848

I also archived the pages and PDFs in that:

ANIMALS ALL DIED IN TESTING OF MRNA VACCINES

When Someone Demands the Official Links To the mRNA Animal Studies Wherein All the Animals Died - Here They Are : CovidVaccineInjury

1st source:

Frontiers | Viral-Induced Enhanced Disease Illness | Microbiology

"Scroll down to 'Coronaviruses Severe Acute Respiratory Syndrome Coronavirus (SARS-CoV) Just a snippet: "Vaccine-induced disease enhancement is also a concern with developing a SARS-CoV vaccine. This was reported in only small subset of SARS-CoV vaccine studies (de Wit et al., 2016). In a mouse study that investigated the role of SARS-CoV vaccine in inducing disease enhancement, Tseng et al. (2012) it was revealed that vaccines were able to protect against SARS-CoV infection, but still induced Th2 directed pulmonary immunopathology suggesting hypersensitivity to SARS-CoV components. In another study, post vaccination challenge of mice with SARS-CoV nucleocapsid protein induced sever pneumonia (Yasui et al., 2008). Likewise, double inactivated SARS-CoV vaccine in mice failed to provide complete protection and caused enhanced eosinophilic pro-inflammatory pulmonary response after infection (Bolles et al., 2011).""

2nd source: In 2004 study caused hepatitis in ferrets:

Immunization with Modified Vaccinia Virus Ankara-Based Recombinant Vaccine against Severe Acute Respiratory Syndrome Is Associated with Enhanced Hepatitis in Ferrets

3rd source: In 2005 mice and civets became sick and more susceptible to coronaviruses after being vaccinated:

Caution raised over SARS vaccine | Nature

Reference 1: Evasion of antibody neutralization in emerging severe acute respiratory syndrome coronaviruses | PNAS

4th source: 2012 study - mice and ferrets developed lung disease:

Immunization with SARS Coronavirus Vaccines Leads to Pulmonary Immunopathology on Challenge with the SARS Virus

5th source: 2016 study of mice once again lung disease:

Full article: Immunization with inactivated Middle East Respiratory Syndrome coronavirus vaccine leads to lung immunopathology on challenge with live virus

"The typical pattern in these studies above is that the children and the animals produced promising antibody responses after being vaccinated. The problem came when the children and animals were exposed to the wild version of the virus. When that happened, an unexplained phenomenon called Antibody Dependent Enhancement (ADE) also known as Vaccine Enhanced Disease (VED) occurred where the immune system produced a "cytokine storm" (i.e. overwhelmingly attacked the body) and the children and animals died."

Caution Urged on SARS Vaccines | Science

"Which is precisely what one of the inventors of the mRNA technology, Dr. Robert Malone has been trying to tell everyone now for months. Numerous attempts have been made to create viral vaccines and all ended in utter failure and all with same pattern - in the 1960's scientists attempted to create a RSV (Respiratory Syncytial Virus) vaccine for infants, in that study they skipped animal trials because they weren't necessary back then, but in the end the vaccinated infants became far more ill than the unvaccinated infants when exposed to the virus in nature, with 80% of the vaccinated infants requiring hospitalization and two of them died."

"Since 2000 there have been many attempts to create coronavirus vaccines and all have ended in failure because the animals in the clinical trials became deathly ill and died, just like the children in the 1960's. I don't what is wrong with you people and why you're all so lazy you can't even be bothered to research and find the studies yourselves- it's not like a debate on some abstract subject, this is your very health and LIFE in the balance. Pathetic."

And here are those PDFs and the archived pages saved to single html files in a 7zip:

[–] • 1 pt

When I try to use the magnifier, it says Access Denied.

[–] • 0 pt

I see. Thank you for noting that. I did not realize basic search functionality was limited, though I expect it is so to prevent abuse.

I would guess that it is likely 'unlocked' upon reaching level 2, which only needs 40 points. You only have 20 comments including this one in the past 3 months your account has existed (1 comment every 4.5 days or so avg.), so you just now hit level 1 with 10 points. Level 2 is reached pretty quickly with a little more participation on the site, though I admit I do not know if it is restricted until level 3 or even 4 but I would not expect it to be so.

[–] • 0 pt

I see, thanks for laying it out for me. I had been wondering about that.

[–] • 2 pts

https://pubmed.ncbi.nlm.nih.gov/?term=antibody+dependent+enhancement

This might start you off, haven't looked through them yet though.

I keep on hearing people reference Merek's in chickens, but I have yet to see a decent citation for it.

Good luck.

[–] • 2 pts

It's marek disease you're talking about https://en.wikipedia.org/wiki/Marek's_disease#Prevention

>Because vaccination does not prevent infection with the virus, Marek's is still transmissible from vaccinated flocks to other birds, including the wild bird population. The first Marek's disease vaccine was introduced in 1970. The disease would cause mild paralysis, with the only identifiable lesions being in neural tissue. Mortality of chickens infected with Marek's disease was quite low. Current strains of Marek virus, decades after the first vaccine was introduced, cause lymphoma formation throughout the chicken's body and mortality rates have reached 100% in unvaccinated chickens. The Marek's disease vaccine is a "leaky vaccine", which means that only the symptoms of the disease are prevented.[12] Infection of the host and the transmission of the virus are not inhibited by the vaccine. This contrasts with most other vaccines, where infection of the host is prevented. Under normal conditions, highly virulent strains of the virus are not selected. A highly virulent strain would kill the host before the virus would have an opportunity to transmit to other potential hosts and replicate. Thus, less virulent strains are selected. These strains are virulent enough to induce symptoms but not enough to kill the host, allowing further transmission. However, the leaky vaccine changes this evolutionary pressure and permits the evolution of highly virulent strains.[13] The vaccine's inability to prevent infection and transmission allows the spread of highly virulent strains among vaccinated chickens. The fitness of the more virulent strains is increased by the vaccine.

[–] • 0 pt

I'm lucky because God hates me enough to not let me die.

#Blessed

[–] • 1 pt

God has a plan for everybody, even for you

And at the end of it you die

[–] • 0 pt

Let me sum them up for you: The entire test group died.

Thats the conclusion of EVERY mRNA trial.

They died, the entire group, every time.

[–] • 0 pt

Here is a recent one: https://www.biorxiv.org/content/10.1101/2020.12.18.423358v1

No peer review yet, and not in-vivo. But they could show in vitro that antibodies created against the "other end" of the spike protein (the one attached to the virus) make it easier for the spike protein to bind to ACE:

Here, we screened a series of anti-spike monoclonal antibodies from COVID-19 patients, and found that some of antibodies against the N-terminal domain (NTD) dramatically enhanced the binding capacity of the spike protein to ACE2, and thus increased SARS-CoV2 infectivity

The common vaccines all create the whole spike protein, including the NTD, so that the protein can fold into its natural shape.

Enhanced binding is one of maybe 5 different forms of ADE. Others are e.g. infection of cells that are not affected if antibodies are not present, destruction of immune system cells and so on.

This could happen with an infection too, not only with vaccination. But there are theories that the quality control (suppressing the bad antibodies and promoting the neutralizing ones) works better in a natural infection compared to the pseudo-infection caused by a vaccine.